江西省白血病与地形地貌类型的关系

通过对地形地貌资料4502个数据,白血病死亡调查资料2239例,研究了白血病死亡率与人群生存区的地形地貌类型（山区、丘陵、岗地、平原）的相关性。结果表明,山区、岗地、平原与白血病死亡率有明显的相关性,等级相关系数分别为-0．1963（P＜0．05）,0．2925（P＜0．0025）,0．3122（P＜0．0025）。     

试说中国陆内构造变形和其地球动力学特征

中国陆内构造变形主要始于晚二叠世中国北方进入后海西地台发展时期。印支期末以至喜马拉雅期,随着特提斯洋的关闭,大陆范围也随即向中国南方和青藏地区增生和扩展。中国的陆内构造变形从后海西地台形成的准平原化阶段即已开始。但规模巨大的变形则发生于燕山构造旋回中期和喜马拉雅旋回。陆内构造变形的规模可分两个等级:一是覆盖整个中国大陆的,另一是局部的。前者两次改变了中国构造-地貌的整体面貌;后者则表现为造山、造盆和微陆块纵向或横向的逃逸,以及由此引起的造山或造盆。根据中国陆内构造变形特征及其与区域构造背景演化的关系分析认为,中国陆内构造变形主要是由周边洲级规模板块运动引起的,同时,随着地壳上部构造变形引发的地壳或岩石圈均衡调整,则使地下深处产生相应的构造响应。     

Mapping the Geographical Diffusion of a Finnish Smallpox Epidemic from Historical Population Records

Smallpox mortality from an early 19th century epidemic in Finland is mapped at the parish village level. First, geographically referenced historical materials are used to construct a map showing the spread of smallpox mortality among villages. Next, the diffusion of smallpox morbidity is simulated by computer for the same set of villages. These two maps are then compared. Results indicate that at this scale maps of mortality diffusion can show general trends but have no direct spatial correspondence to the underlying pattern of morbidity diffusion. Mortality maps cannot be used as a surrogate measure of infectious contact behavior at micro scales of analysis.     

基于抗震性态的设防标准研究

以基于性态的抗震设计为出发点,并以我国为例,针对目前国内外抗震设防标准的若干问题和不足,提出了基于性态抗震设计的三环节抗震设防方法．将现有的设防内容改为确定结构的抗震设计类别、确定设计烈度或设计地震动参数、确定建筑的重要性等级３个方面,并分别在每一方面进行具体深入的研究,从而形成了一套完整的基于性态的抗震设防标准确定的原则、方法和框架,可以直接用来编制规范供抗震设计使用．      

利用卫星光谱分析技术进行断层逸出气体监测

在不少震例中,临震前地壳出现气体异常,震前地下岩石受力产生裂隙,隐藏在地层中的CO2、CH4等温室气体逸出地表或水面。利用卫星光谱分析技术对断层逸出气体组分含量和总量进行监测,筛选出能够反应地震孕育过程中地下逸出气体异常演化特征的参数,建立地震短期预测的动态演化方法。对于认识震前地表气体异常机理,了解流体震兆的演变规律与这些气体异常的动态关系,改善卫星遥感地震预测,是很必要的。     

Tectonic elements controlling the evolution of the Gulf of Saros (northeastern Aegean Sea, Turkey)

Tectonic elements controlling the evolution of the Gulf of Saros have been studied based upon the high-resolution shallow seismic data integrated with the geological field observations. Evolution of the Gulf of Saros started in the Middle to Late Miocene due to the NW–SE compression caused by the counterclockwise movement of the Thrace and Biga peninsulas along the Thrace Fault Zone. Hence, the North Anatolian Fault Zone is not an active structural element responsible for the starting of the evolution of the Gulf of Saros. The compression caused by the rotational movement was compensated by tectonic escape along the pre-existing Ganos Fault System. Two most significant controllers of this deformation are the sinistral Ganos Fault and the dextral northern Saros Fault Zone both extending along the Gulf of Saros. The most important evidences of this movement are the left- and right-oriented shear deformations, which are correlated with structural elements, observed on the land and on the high-resolution shallow seismic records at the sea. Another important line of evidence supporting the evolution of this deformation is that the transgression started in the early-Late Miocene and turned, as a result of regional uplift, into a regression on the Gelibolu Peninsula during the Turolian and in the north of the Saros Trough during the Early Pliocene. The deformation on the Gelibolu Peninsula continued effectively until the Pleistocene. Taking into account the fact that this deformation affected the Late Pleistocene units of the Marmara Formation, the graben formation of the Gulf of Saros is interpreted as a Recent event. However, at least a small amount of compression on the Gelibolu Peninsula is observed. It is also evident that compression ceased at the northern shelf area of the Gulf of Saros.     

The impact of anoxia on pelagic macrofauna during the Toarcian Oceanic Anoxic Event (Early Jurassic)

Extreme environmental change during the Toarcian Oceanic Anoxic Event had widespread impacts on marine biota. This study provides new evidence, from the Yorkshire coast sections, UK, that the event was associated with periods of elevated fish and ammonite mortality. Using a synthesis of pelagic macrofaunal changes, benthic macrofaunal data and geochemical proxies we show that there are stratigraphical correlations between: (1) pelagic macrofaunal ranges and abundance, (2) benthic macrofaunal abundance, and (3) geochemical proxies that indicate deoxygenation. We identify eight stratigraphical intervals of differing character. Results suggest two major phases of relatively persistent deoxygenation with photic zone euxinia. The cyclostratigraphic timescale indicates that each phase lasted at least tens of thousands of years. Belemnite migration during the event probably resulted from increased seawater temperatures and low food supply similar to that observed for many marine taxa, including squid, within the present-day oceans.     

中国人口死亡率空间格局演变及其影响因素

人口死亡率是人口转型关键因素之一,而人口转型又关系着地区社会经济发展质量。以2000—2015年粗死亡率数据为基础,通过变异系数、趋势分析和空间自相关分析中国人口死亡率空间格局差异及演变态势,并结合偏最小二乘回归定量分析人口死亡率影响因素。结果发现：（1）中国人口死亡率大致呈西北、东北和东南低,中部及西南高态势,且各时段人口死亡率降低省份高于增长省份;（2）中国各省死亡率变异系数差异悬殊,且在空间趋势中东西方向呈现两端低中间高、南北方向由两端低中间高向北低南高转变;（3）人口死亡率全局空间集聚程度不断减弱,区域差异缩小,且局部空间上以高-高和低-低聚集为主;（4）人口老龄化是人口死亡率提升的主要推动因子,其次分别为工业废水排放总量、突发环境事件次数和气候条件等。人均生产总值对人口死亡率起主要抑制作用,其次分别为每千人口床位数、每十万人口大学平均在校生数、人均用水量和空气质量。农作物受灾面积合计对人口死亡率在不同年份作用力不一致。     

PM2.5污染对中国人口死亡率的影响——基于346个城市面板数据的实证分析

中国生态文明建设和“健康中国”战略强调切实治理影响人口健康的环境问题,建设健康人居环境。论文基于2000年和2010年中国人口普查资料以及2005年和2015年各省级行政单元1%人口抽样调查等数据资料,论文使用探索性空间分析方法刻画中国城市人口死亡率的时空变化特征,并采用空间回归方法,揭示城市PM_2.5的平均浓度对人口死亡率的影响及其空间溢出效应,以及社会经济因素对PM_2.5—人口死亡率关联的调节效应。结果表明：① 中国城市人口死亡率的空间分布特征呈现明显的异质性,高死亡率地区早期集聚分布于西南地区,2005年后在西南地区、华北地区、华东地区和华中地区呈现逐渐集聚分布态势。低死亡率地区长期集中分布于西北地区、东北地区、长三角地区、珠三角地区和京津两市。② 人口死亡率的分布存在空间关联性,高—高类型地区早期集中分布于西南地区,后期向东扩展;低—低类型地区主要分布于北疆、内蒙古西部和广东省及其周边地区。③ 城市PM_2.5浓度对人口死亡率具有显著的正向影响,并且对邻近地区的人口死亡率具有显著的空间溢出效应。④ 中国城市PM_2.5浓度对人口死亡率的影响存在学历差异和城乡差异,地区高学历人群集聚可降低PM_2.5的健康风险,城镇化发展进程缓慢则会加重PM_2.5的健康风险。研究旨在为防范空气污染暴露导致的健康风险、建设健康人居环境提供科学依据。     

Aeronomy of extra-solar giant planets at small orbital distances

One-dimensional aeronomical calculations of the atmospheric structure of extra-solar giant planets in orbits with semi-major axes from 0.01 to 0.1 AU show that the thermospheres are heated to over 10,000 K by the EUV flux from the central star. The high temperatures cause the atmosphere to escape rapidly, implying that the upper thermosphere is cooled primarily by adiabatic expansion. The lower thermosphere is cooled primarily by radiative emissions from H⁺₃, created by photoionization of H₂ and subsequent ion chemistry. Thermal decomposition of H₂ causes an abrupt change in the composition, from molecular to atomic, near the base of the thermosphere. The composition of the upper thermosphere is determined by the balance between photoionization, advection, and H⁺ recombination. Molecular diffusion and thermal conduction are of minor importance, in part because of large atmospheric scale heights. The energy-limited atmospheric escape rate is approximately proportional to the stellar EUV flux. Although escape rates are large, the atmospheres are stable over time scales of billions of years.