A NEW MODEL FOR PEBBLE BEACH TRACER DISPERSAL

A series of eight tracer experiments were carried out over one year (1983) on a low energy, macro-tidal beach in South Wales, UK. Tracer movement was monitored and 30 pebbles adjacent to coloured tracer pebbles were measured. Measurements were of the A, B, and C axes, roundness, Maximum Projection Sphericity and Oblate-Prolate indices. Analysis shows that through time, the percentage rejection of these properties increased. It is theorised that the surface tracers are 'misfits' or 'rejects' which have not found their equilibrium position in the pebble facies.     

沿海城市旅游业的SWOT分析和发展战略探讨——以大连市为例

文章结合大连市旅游业发展的实际，对大连市旅游业进行了SWOT分析，在此基础上提出了未来大连市旅游业的发展目标及应达到的相应标准，并为实现和达到这些目标提出了旅游业的管理、宣传、人才培养等多方面的相关举措。     

西南太平洋彭林海盆表层和埋藏锰结核成分和生长史

本文所研究的样品由《Hakurei—maru》号科学考察船进行的GH80—1和GH83—3两个航次所采集．这两次航行的目的是作为日本“中太平洋锰结核调查”五年计划(1979—1983)任务的一部分，在威克-塔希提横断层上进行地质调查(GSJ)。     

无机碳与雨生红球藻(Haematococcus pluvialis)细胞调节物质

以单细胞雨生红球藻为材料，采用酸碱滴定和CO2加富通气培养微藻的方法，对旧液中HCO3^-和CO3^2-浓度变化以及对红球藻细胞生长的影响进行了研究。结果表明，旧液具有限制红球藻细胞生长和诱导细胞转化的作用。同时，旧液中无机碳离子浓度明显高于新液。培养液中富含CO3^2-时，各细胞数量与CO3^2-浓度呈正相关，相关系数为0．88。溶液中仅有HCO3^-时，各细胞数与HCO3^-浓度也呈正相关性。因此，排除了CO3^2-和HCO3^-作为旧液中的调节物质，限制红球藻细胞生长和诱导细胞转化的可能性。旧液乙酸乙酯提取物生物检测实验表明，在粗提取物中有降低细胞增长和诱导细胞转化的活性，表明调节物质能溶于有机相，也反过来证实无机碳离子不是旧液中的调节物质。DNA含量和倍性分析结果表明，红球藻游动细胞DNA复制可以加倍后不经过原生质分裂就可以再次进行，因此推测旧液中的调节物质对原生质分裂过程产生抑制作用，而不对DNA复制过程产生抑制作用。     

由偏顶蛤贝壳中稳定同位素剖面追溯海水温度记录

本文用在加拿大诺瓦·斯科舍省芬克湾采集到的两只活体偏顶蛤(Modiolus)贝壳上的氧同位素组成重建了过去十年间的底层海水温度记录,它也可作为一种独立的方法来确定贝壳上外生长线的沉积时间。由于对贝壳进行了密集采样(可多至每个贝壳沉积年份取15个样品),贝壳上的δ~(16)O变化反映了芬克湾底层海水的季节性变化。两只贝壳所得的温度剖面符合得很好,说明它们基本上是与真实的环境温度相对应的。本研究证明在海胆疾病爆发时底层海水的温度并没有异常升高,这说明疾病传播的关键因素是带病的生物体,而不是温度。同位素技术也说明了在贝壳外表面上可见的外生长线是年轮性的,它们是在每年冬季最低水温时沉积出来的。     

Population structure of Haliotis rubra from South Australia inferred from nuclear and mtDNA analyses

1Introduction ThemajorityofAustralia’sabalonefisheryex ports(5.135kt,worth＄216millionin2002～2003,ABARE2004)consistofblacklipabalone(HaliotisrubraLeach,1814).AssuchH.rubrais consideredasanimportantmarineresourcewithin Australia.Likemanyabalonespecieswor…     

Bacterial killing by Mytilus hemocyte monolayers as a model for investigating the signaling pathways involved in mussel immune defence

The signaling pathways involved in mussel immune defence were investigated utilizing a model of killing of Escherichia coli by Mytilus galloprovincialis hemocytes in a co-culture setting. In particular, the role played by different mitogen activated protein kinases (MAPKs) and by the production of eicosanoids were investigated utilising specific cell permeant, pharmacological enzyme inhibitors. Hemocyte pretreatment with the p38 MAPK inhibitor SB203580 significantly reduced bacterial killing, whereas PD98059 (an inhibitor of ERK--extracellularly regulated kinase--MAPK activation) had no significant effect. Wortmannin also inhibited bacterial killing, indicating a crucial role for PI3-kinase activation in the immune response. Killing of E. coli was also reduced by inhibitors of both PLA2 and cyclooxygenase activities, indicating that eicosanoid production is involved in mediating the response to bacterial challenge. The results demonstrate that bacterial killing by mussel hemocytes is particularly sensitive to inhibitors of the key steps involved in the transduction of bacterial signals into the host cell. Moreover, these data indicate that the hemocyte bactericidal activity can be suitably utilized not only for identifying the signaling pathways involved in the response to bacterial infection, but also as a potential investigative-toxicology model to test drugs and contaminants for their effect on the overall mussel immune defence.     

Can turbidity caused by Corophium volutator (Pallas) activity be used to assess sediment toxicity rapidly?

The standard toxicity test organism, Corophium volutator, exhibits a behavioural response to contaminated sediments that causes increased turbidity of overlying water. We quantify the effects of this response to an estuarine sediment spiked with copper and hydrocarbon contaminated sediments from an oil installation in the North Sea. Turbidity measured 24 h after the start of a toxicity test shows a strong relationship with contaminant concentrations and with mortality after 10 days. Turbidity measurements can therefore give a rapid indication of sediment toxicity, permitting a reduction in storage time of sediments to be used in dilution series and toxicity identification evaluation (TIE) tests, reducing the likelihood of contaminants degrading prior to testing.